Dissociative disorder
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Dissociative disorders (DD) are conditions that involve significant disruptions and/or breakdowns "in the normal integration of consciousness, memory, identity, emotion, perception, body representation, motor control, and behavior." People with dissociative disorders also use dissociation as a defense mechanism involuntarily. The individual experiences these dissociations to protect themselves from traumatic stress. Some dissociative disorders are triggered by significant psychological trauma, though depersonalization-derealization disorder may be preceded by lesser stress, psychoactive substances, or no identifiable trigger at all.[1]

The dissociative disorders listed in the American Psychiatric Association's DSM-5 are as follows:[2]

The ICD-11 lists dissociative disorders as:[7]

Causes and treatment

Dissociative disorders most often develop as a way to cope with psychological trauma. People with dissociative disorders were commonly subjected to chronic physical, sexual, or emotional abuse as children (or, less frequently, an otherwise frightening or highly unpredictable home environment). Some categories of DD, however, can form due to trauma that occurs later in life and is unrelated to abuse, such as war or the death of a loved one.

Dissociative disorders, especially dissociative identity disorder (DID), should not be treated with an extraordinary or supernatural status. DDs would be better examined and treated through the lens of any other psychological disorder.[8][clarification needed]

Dissociative identity disorder

Cause: Dissociative identity disorder is caused by ongoing childhood trauma that occurs before the ages of six to nine.[9][10] People with dissociative identity disorder usually have close relatives who have also had similar experiences.[11]

Treatment: Long-term psychotherapy to improve the patient's quality of life. Psychotherapy often involves hypnosis (to help a patient remember and work through the trauma), creative art therapy (using creative process to help a person who cannot express their thoughts), cognitive therapy (talk therapy to identify unhealthy and negative beliefs or behaviors), and medications (antidepressants, anti-anxiety medications, or sedatives). These medications can help control the symptoms associated with DID and other DD, but there are no medications yet that specifically treat dissociative disorders.[12]

Dissociative amnesia

Cause: Psychological trauma. While a history of child abuse is common in patients, it is not a necessary factor in determining if a person will develop dissociative amnesia.[13]

Treatment: Psychotherapy counseling or psychosocial therapy which involves talking about the disorder and related issues with a mental health provider. The medication pentothal can sometimes help to restore the memories.[11] The length of an event of dissociative amnesia may be a few minutes or several years. If an episode is associated with a traumatic event, the amnesia may clear up when the person is removed from the traumatic situation.

Depersonalization-derealization disorder

Cause: While not as strongly linked as other dissociative disorders, there is a correlation between depersonalization-derealization disorder and childhood trauma, especially emotional abuse or neglect. It can also be caused by other forms of stress such as sudden death of a loved one.[14]

Treatment: Same treatment as dissociative amnesia. An episode of depersonalization-derealization disorder can be as brief as a few seconds or continue for several years.[11]

Neuroscience of dissociative disorders

Differences in brain activity

Dissociative disorders are characterized by distinct brain differences in the activation of various brain regions including the inferior parietal lobe, prefrontal cortex, and limbic system.[15]

Those with dissociative disorders have higher activity levels in the prefrontal lobe and a more inhibited limbic system on average than healthy controls.[15] Heightened corticolimbic inhibition is associated with distinctly dissociative symptoms such as depersonalization and derealization.[15] The function of these symptoms is thought to be a coping mechanism employed in extremely threatening or traumatic events.[16] By inhibiting structures in the limbic system, such as the amygdala, the brain is able to reduce extreme levels of arousal.[15] In the dissociative subtype of PTSD, there is both excessive control of emotions through suppressed limbic structures and insufficient control of emotions in the hyperactivity of the medial prefrontal cortex. Increased activity in the medial prefrontal cortex is associated with non-dissociative symptoms such as re-experiencing and hyperarousal.[15]

Differences in volume of brain structures

There are notable differences in the volume of certain areas of the brain such as reduced cortical and subcortical volumes in the hippocampus and amygdala.[17] Reduced volume of the amygdala may account for the lessened emotional reactivity observed during dissociation.[17] The hippocampus is associated with learning and the formation of memory, and its reduced volume is associated with impairments in memory for those with DID and PTSD.[18] Brain-imaging studies demonstrating the link between reduced hippocampal volume and DID as well as PTSD have added to empirical support for the controversial existence of the disorder, as additional brain-imaging studies have demonstrated a negative correlation between hippocampal volume and early childhood trauma (which is hypothesized to be a potential etiological factor for dissociative symptoms).[19][20][21]

The Amygdala
The Hippocampus
The Prefrontal Cortex

Medications

There are no medications to cure or completely treat dissociative disorders, however, drugs to treat anxiety and depression that may accompany the disorders can be given.[22]

Diagnosis and prevalence

The lifetime prevalence of dissociative disorders varies from 10% in the general population to 46% in psychiatric inpatients.[23] Diagnosis can be made with the help of structured clinical interviews such as the Dissociative Disorders Interview Schedule (DDIS) and the Structured Clinical Interview for DSM-IV Dissociative Disorders (SCID-D-R), and behavioral observation of dissociative signs during the interview.[23][24] Additional information can be helpful in diagnosis, including the Dissociative Experiences Scale or other questionnaires, performance-based measures, records from doctors or academic records, and information from partners, parents, or friends.[24] A dissociative disorder cannot be ruled out in a single session and it is common for patients diagnosed with a dissociative disorder to not have a previous dissociative disorder diagnosis due to a lack of clinician training.[24] Some diagnostic tests have also been adapted or developed for use with children and adolescents such as the Adolescent Dissociative Experiences Scale,[25] Children's Version of the Response Evaluation Measure (REM-Y-71), Child Interview for Subjective Dissociative Experiences, Child Dissociative Checklist (CDC), Child Behavior Checklist (CBCL) Dissociation Subscale, and the Trauma Symptom Checklist for Children Dissociation Subscale.[26]

Dissociative disorders have been found to be quite prevalent in outpatient populations, as well as within low-income communities. One study found that in a population of poor inner-city outpatients, there was a 29% prevalence of dissociative disorders.[27]

There are problems with classification, diagnosis and therapeutic strategies of dissociative and conversion disorders which can be understood by the historic context of hysteria. Even current systems used to diagnose DD such as the DSM-IV and ICD-10 differ in the way the classification is determined.[28] In most cases mental health professionals are still hesitant to diagnose patients with Dissociative Disorder, because before they are considered to be diagnosed with Dissociative Disorder these patients have more than likely been diagnosed with major depressive disorder, anxiety disorder, and most often post-traumatic stress disorder.[29] It has been found from interviews with those who may be afflicted with dissociative disorders may be more effective at getting an accurate diagnosis than self-scoring assessments and scales.[27]

The prevalence of dissociative disorders is not completely understood due to the many difficulties in diagnosing dissociative disorders. Many of these difficulties stem from a misunderstanding of dissociative disorders, from an unfamiliarity diagnosis or symptoms to disbelief in some dissociative disorders entirely.[30] Due to this it has been found that only 28% to 48% of people diagnosed with a dissociative disorder receive treatment for their mental health.[31] Patients who are misdiagnosed are often those more likely to be hospitalised repeatedly, and lack of treatment can result in intensive outpatient treatment and higher rates of disability.[31]

An important concern in the diagnosis of dissociative disorders in forensic interviews is the possibility that the patient may be feigning symptoms in order to escape negative consequences. Young criminal offenders report much higher levels of dissociative disorders, such as amnesia. In one study it was found that 1% of young offenders reported complete amnesia for a violent crime, while 19% claimed partial amnesia.[32] There have also been cases in which people with dissociative identity disorder provide conflicting testimonies in court, depending on the personality that is present.[33][better source needed] The world-wide prevalence of dissociative disorders is not well understood due to different cultural beliefs surrounding human emotions and the human brain.[34]

Children and adolescents

Dissociative disorders (DD) are widely believed to have roots in adverse childhood experiences including abuse and loss, but the symptoms often go unrecognized or are misdiagnosed in children and adolescents.[26][35][36][verification needed] However, a recent western Chinese study showed an increase in awareness of dissociative disorders present in children.[37] These studies show that DD's have an intricate relationship with the patient's mental, physical and socio-cultural environments.[37] This study suggested that dissociative disorders are more common in Western, or developing countries,[37] however, some cases have been seen in both clinical and non-clinical Chinese populations.[37] There are several reasons why recognizing symptoms of dissociation in children is challenging: it may be difficult for children to describe their internal experiences; caregivers may miss signals or attempt to conceal their own abusive or neglectful behaviors;[citation needed] symptoms can be subtle or fleeting;[26] disturbances of memory, mood, or concentration associated with dissociation may be misinterpreted as symptoms of other disorders.[26]

Another resource, Beacon House, informs us of dissociative disorder in children, suggesting that it is a survival mechanism that often goes unnoticed in children that have been traumatised.[38] Dr. Shoshanah Lyons suggests that traumatised children often continue to dissociate even though they might not be in any danger, and that they are often unaware that they are dissociating.[38] In addition to developing diagnostic tests for children and adolescents (see above), a number of approaches have been developed to improve recognition and understanding of dissociation in children. Recent research has focused on clarifying the neurological basis of symptoms associated with dissociation by studying neurochemical, functional and structural brain abnormalities that can result from childhood trauma.[35] Others in the field have argued that recognizing disorganized attachment (DA) in children can help alert clinicians to the possibility of dissociative disorders.[36] In their 2008 article, Rebecca Seligman and Laurence Kirmayer suggest the existence of evidence of linkages between trauma experienced in childhood and the capacity for dissociation or depersonalisation.[39] They also suggest that individuals who are able to utilise dissociative techniques are able to keep this as an extended strategy to cope with stressful situations.[39]

Clinicians and researchers[who?] stress the importance of using a developmental model to understand both symptoms and the future course of DDs.[26][35] In other words, symptoms of dissociation may manifest differently at different stages of child and adolescent development and individuals may be more or less susceptible to developing dissociative symptoms at different ages. Further research into the manifestation of dissociative symptoms and vulnerability throughout development is needed.[26][35] Related to this developmental approach, more research is required to establish whether a young patient's recovery will remain stable over time.[40]

Current debates and the DSM-5

A number of controversies surround DD in adults as well as children. First, there is ongoing debate surrounding the etiology of dissociative identity disorder (DID). The crux of this debate is if DID is the result of childhood trauma and disorganized attachment.[35][41] A proposed view is that dissociation has a physiological basis, in that it involves automatically triggered mechanisms such as increased blood pressure and alertness, that would, as Lynn contends, imply its existence as a cross-species disorder.[42] A second area of controversy surrounds the question of whether or not dissociation as a defense versus pathological dissociation are qualitatively or quantitatively different. Experiences and symptoms of dissociation can range from the more mundane to those associated with posttraumatic stress disorder (PTSD) or acute stress disorder (ASD) to dissociative disorders.[26] Mirroring this complexity, the DSM-5 workgroup considered grouping dissociative disorders with other trauma/stress disorders,[43] but instead decided to put them in the following chapter to emphasize the close relationship.[44] The DSM-5 also introduced a dissociative subtype of PTSD.[44]

A 2012 review article supports the hypothesis that current or recent trauma may affect an individual's assessment of the more distant past, changing the experience of the past and resulting in dissociative states.[45] However, experimental research in cognitive science continues to challenge claims concerning the validity of the dissociation construct, which is still based on Janetian notions of structural dissociation.[4][46] Even the claimed etiological link between trauma/abuse and dissociation has been questioned. Links observed between trauma/abuse and DD are largely only present from a Western cultural context. For non-Western cultures dissociation "may constitute a "normal" psychological capacity".[citation needed] An alternative model proposes a perspective on dissociation based on a recently established link between a labile sleep–wake cycle and memory errors, cognitive failures, problems in attentional control, and difficulties in distinguishing fantasy from reality."[47]

Debates around DD also stem from Western versus non-Western lenses of viewing the disorder, and associated views of causes of DD. DID was initially believed to be specific to the West, until cross-cultural studies indicated its occurrence worldwide.[42] Conversely, anthropologists have largely done little work on DD in the West relating to its perceptions of possession syndromes that would be present in non-Western societies.[citation needed] While dissociation has been viewed and catalogued by anthropologists differently in the West and non-Western societies, there are aspects of each that show DD has universal characteristics. For example, while shamanic and rituals of non-Western societies may hold dissociative aspects, this is not exclusive as many Christian sects, such as "possession by the Holy Ghost" share similar qualities to those of non-Western trances.[citation needed]

See also

References

  1. ^ Simeon, D; Abugel, J (2006). Feeling Unreal: Depersonalization Disorder and the Loss of the Self. New York, NY: Oxford University Press. p. 17. ISBN 0195170229. OCLC 61123091.
  2. ^ a b Diagnostic and statistical manual of mental disorders : DSM-5 (5th ed.). Arlington, VA: American Psychiatric Association. 2013. pp. 291–307. ISBN 9780890425541.
  3. ^ Schacter, D. L., Gilbert, D. T., & Wegner, D.M. (2011). Psychology: Second Edition, pages 572-573 New York, NY: Worth.
  4. ^ a b Maldonado, R.J.; Spiegel, D. (2019). "Dissociative Disorders". In Weiss Roberts, Laura; Hales, Robert E.; Yudofsky, Stuart C. (eds.). The American Psychiatric Publishing Board Review Guide for Psychiatry (7th ed.). American Psychiatric Pub. ISBN 978-1-61537-150-1.
  5. ^ First, M. B., Kay, J., Lieberman, J. A., Riba, M. B., Tasman, A., eds. Psychiatry. 4th ed. Chichester, UK: John Wiley & Sons; 2015. p1187. ISBN 978-1-118-84547-9
  6. ^ Diagnostic and statistical manual of mental disorders : DSM-5 (5th ed.). Arlington, VA: American Psychiatric Association. 2013. p. 812. ISBN 9780890425541.
  7. ^ "ICD-11 - Mortality and Morbidity Statistics". icd.who.int.
  8. ^ Deeley, P. Q (2003). "Social, Cognitive, and Neural Constraints on Subjectivity and Agency: Implications for Dissociative Identity Disorder". Philosophy, Psychiatry, & Psychology. 10 (2): 161–167. doi:10.1353/ppp.2003.0095. S2CID 145372363.
  9. ^ Spigel, David; et al. "Dissociative disorders in DSM5DMS". Retrieved 3 January 2018.
  10. ^ Salter, Micahel; Dorahy, Martin; Middleton, Warwick (4 October 2017). "Dissociative identity disorder exists and is the result of childhood trauma". The Conversation. Retrieved 3 January 2018.
  11. ^ a b c Miller, John L. (3 February 2014). "Dissociative Disorders". athealth.com. Retrieved 14 December 2016.
  12. ^ "Dissociative disorders: Treatments and drugs". Mayo Clinic. 3 March 2011. Archived from the original on 22 October 2011.
  13. ^ Diagnostic and statistical manual of mental disorders: DSM-5 (5th ed.). Arlington, VA: American Psychiatric Association. 2013. p. 299. ISBN 9780890425541.
  14. ^ Diagnostic and statistical manual of mental disorders: DSM-5 (5th ed.). Arlington, VA: American Psychiatric Association. 2013. p. 304. ISBN 9780890425541.
  15. ^ a b c d e Spiegel, David; Lewis-Fernández, Roberto; Lanius, Ruth; Vermetten, Eric; Simeon, Daphne; Friedman, Matthew (28 March 2013). "Dissociative Disorders in DSM-5". Annual Review of Clinical Psychology. 9 (1): 299–326. doi:10.1146/annurev-clinpsy-050212-185531. ISSN 1548-5943.
  16. ^ Diagnostic and statistical manual of mental disorders: DSM-IV-TR (4. ed., text rev., 13. print ed.). Arlington, VA: American Psychiatric Assoc. 2009. ISBN 978-0-89042-024-9.
  17. ^ a b Lotfinia, Shahab; Soorgi, Zohre; Mertens, Yoki; Daniels, Judith (September 2020). "Structural and functional brain alterations in psychiatric patients with dissociative experiences: A systematic review of magnetic resonance imaging studies". Journal of Psychiatric Research. 128: 5–15. doi:10.1016/j.jpsychires.2020.05.006.
  18. ^ Blihar, David; Delgado, Elliott; Buryak, Marina; Gonzalez, Michael; Waechter, Randall (September 2020). "A systematic review of the neuroanatomy of dissociative identity disorder". European Journal of Trauma & Dissociation. 4 (3): 100148. doi:10.1016/j.ejtd.2020.100148.
  19. ^ Reinders, A. A. T. S.; Chalavi, S.; Schlumpf, Y. R.; Vissia, E. M.; Nijenhuis, E. R. S.; Jäncke, L.; Veltman, D. J.; Ecker, C. (February 2018). "Neurodevelopmental origins of abnormal cortical morphology in dissociative identity disorder". Acta Psychiatrica Scandinavica. 137 (2): 157–170. doi:10.1111/acps.12839. ISSN 0001-690X.
  20. ^ Chalavi, Sima; Vissia, Eline M.; Giesen, Mechteld E.; Nijenhuis, Ellert R.S.; Draijer, Nel; Cole, James H.; Dazzan, Paola; Pariante, Carmine M.; Madsen, Sarah K.; Rajagopalan, Priya; Thompson, Paul M.; Toga, Arthur W.; Veltman, Dick J.; Reinders, Antje A.T.S. (May 2015). "Abnormal hippocampal morphology in dissociative identity disorder and post‐traumatic stress disorder correlates with childhood trauma and dissociative symptoms". Human Brain Mapping. 36 (5): 1692–1704. doi:10.1002/hbm.22730. ISSN 1065-9471. PMC 4400262.
  21. ^ Blihar, David; Crisafio, Anthony; Delgado, Elliott; Buryak, Marina; Gonzalez, Michael; Waechter, Randall (27 May 2021). "A Meta-Analysis of Hippocampal and Amygdala Volumes in Patients Diagnosed With Dissociative Identity Disorder". Journal of Trauma & Dissociation. 22 (3): 365–377. doi:10.1080/15299732.2020.1869650. ISSN 1529-9732.
  22. ^ "What is dissociation and dissociative identity disorder (DID)?". What are the signs and symptoms of dissociation and dissociative disorder?.
  23. ^ a b Ross; et al. (2002). "Prevalence, Reliability and Validity of Dissociative Disorders in an Inpatient Setting". Journal of Trauma & Dissociation. 3: 7–17. doi:10.1300/J229v03n01_02. S2CID 144490486.
  24. ^ a b c Bailey, Tyson D.; Boyer, Stacey M.; Brand, Bethany L. (2019). "Dissociative Disorders". In Segal, Daniel L. (ed.). Diagnostic Interviewing (5th ed.). Springer. ISBN 978-1-4939-9127-3.
  25. ^ "Guidelines for the Evaluation and Treatment of Dissociative Symptoms in Children and Adolescents: International Society for the Study of Dissociation" (PDF). Journal of Trauma & Dissociation. 5 (3): 119–150. 4 October 2004. doi:10.1300/J229v05n03_09. ISSN 1529-9732. S2CID 220430260.
  26. ^ a b c d e f g Steiner, H.; Carrion, V.; Plattner, B.; Koopman, C. (2002). "Dissociative symptoms in posttraumatic stress disorder: diagnosis and treatment". Child and Adolescent Psychiatric Clinics of North America. 12 (2): 231–249. doi:10.1016/s1056-4993(02)00103-7. PMID 12725010.
  27. ^ a b Foote, B; et al. (2006). "Prevalence of Dissociative Disorders in Psychiatric Outpatients". American Journal of Psychiatry. 4 (163): 623–629. doi:10.1176/ajp.2006.163.4.623. PMID 16585436.
  28. ^ Splitzer, C; Freyberger, H.J. (2007). "Dissoziative Störungen (Konversionsstörungen)". Psychotherapeut.
  29. ^ [Nolen-Hoeksema, S. (2014). Somatic Symptom and Dissociative Disorders. In (ab)normal Psychology (6th ed., p. 164). Penn, Plaza, New York: McGraw-Hill.]
  30. ^ Coons, P. M (1998). "The Dissociative Disorders: Rarely Considered and Underdiagnosed". Psychiatric Clinics of North America. 3 (21): 637–648. doi:10.1016/S0193-953X(05)70028-9. PMID 9774801.
  31. ^ a b Nester, M. S; Hawkins, S. L; Brand, B. L (2022). "Barriers to accessing and continuing mental health treatment among individuals with dissociative symptoms". European Journal of Psychotraumatology. 1 (13). doi:10.1080/20008198.2022.2031594. PMC 8856065. PMID 35186217.
  32. ^ Evans, Ceri; Mezey, Gillian; Ehlers, Anke (2009). "Amnesia for violent crime among young offenders". Journal of Forensic Psychiatry & Psychology. 20 (1): 85–106. doi:10.1080/14789940802234471. PMC 2720170. PMID 19668341.
  33. ^ Haley, J. (2003). "Defendant's wife testifies about his multiple personas". Bellingham Herald: B4.
  34. ^ Seligman, R; Brown, R. A (2010). "Theory and method at the intersection of anthropology and cultural neuroscience". Social Cognitive and Affective Neuroscience. 2 (5): 130–137. doi:10.1093/scan/nsp032. PMC 2894668. PMID 19965815.
  35. ^ a b c d e Diseth, T. (2005). "Dissociation in children and adolescents as reaction to trauma - an overview of conceptual issues and neurobiological factors". Nordic Journal of Psychiatry. 59 (2): 79–91. doi:10.1080/08039480510022963. PMID 16195104. S2CID 25581805.
  36. ^ a b Waters, F. (July–August 2005). "Recognizing dissociation in preschool children". The International Society for the Study of Dissociation News. 23 (4): 1–4.
  37. ^ a b c d Fang, Z; et al. (2021). "Characteristics and outcomes of children with dissociative (conversation) disorders in western China: a retrospective study". BMC Psychiatry. 21 (1): 31. doi:10.1186/s12888-021-03045-0. PMC 7802240. PMID 33435924.
  38. ^ a b Lyons, S. "Dissociation in Children and Teens". Beacon House Therapeutic Services & Trauma Team.
  39. ^ a b Seligman, R; Kirmayer, LJ (2008). "Dissociative Experience and Cultural Neuroscience: Narrative, Metaphor and Mechanism". Culture, Medicine and Psychiatry. 32 (1): 31–64. doi:10.1007/s11013-007-9077-8. PMC 5156567. PMID 18213511.
  40. ^ Jans, Thomas; Schneck-Seif, Stefanie; Weigand, Tobias; Schneider, Wolfgang; Ellgring, Heiner; Wewetzer, Christoph; Warnke, Andreas (2008). "Long-term outcome and prognosis of dissociative disorder with onset in childhood or adolescence". Child and Adolescent Psychiatry and Mental Health. 2 (1): 19. doi:10.1186/1753-2000-2-19. PMC 2517058. PMID 18651951.
  41. ^ Boysen, Guy A. (2011). "The Scientific Status of Childhood Dissociative Identity Disorder: A Review of Published Research". Psychotherapy and Psychosomatics. 80 (6): 329–34. doi:10.1159/000323403. PMID 21829044. S2CID 6083787.
  42. ^ a b Lynn, C. D (2005). "Adaptive and Maladaptive Dissociation: An Epidemiological and Anthropological Comparison and Proposition for an Expanded Dissociation Model". Anthropology of Consciousness. 2 (61): 16–49. doi:10.1525/ac.2005.16.2.16.
  43. ^ Brand, Bethany L.; Lanius, Ruth; Vermetten, Eric; Loewenstein, Richard J.; Spiegel, David (2012). "Where Are We Going? An Update on Assessment, Treatment, and Neurobiological Research in Dissociative Disorders as We Move Toward the DSM-5". Journal of Trauma & Dissociation. 13 (1): 9–31. doi:10.1080/15299732.2011.620687. PMID 22211439. S2CID 8204753.
  44. ^ a b American Psychiatric Association (2013). Diagnostic and Statistical Manual of Mental Disorders (DSM-5®) (5th ed.). American Psychiatric Pub. pp. 528–556. ISBN 978-0-89042-557-2.
  45. ^ Stern DB (January 2012). "Witnessing across time: accessing the present from the past and the past from the present". The Psychoanalytic Quarterly. 81 (1): 53–81. doi:10.1002/j.2167-4086.2012.tb00485.x. PMID 22423434. S2CID 5728941.
  46. ^ Heim, Gerhard; Bühler, Karl-Ernst (3 April 2019). Craparo, Giuseppe; Ortu, Francesca; van der Hart, Onno (eds.). Pierre Janet's views on the etiology, pathogenesis, and therapy of dissociative disorders 1 (1 ed.). Routledge. pp. 178–199. doi:10.4324/9780429201875-14. ISBN 978-0-429-20187-5. S2CID 146072691. Retrieved 24 July 2020. ((cite book)): |work= ignored (help)
  47. ^ Lynn, SJ; et al. (2012). "Dissociation and dissociative disorders: challenging conventional wisdom". Current Directions in Psychological Science. 21 (1): 48–53. doi:10.1177/0963721411429457. S2CID 4495728.

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